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Such a severely altered mineralization structure inevitably leads to unusual compliance, other aberrant biomechanical properties, and altered remodeling of bone tissue, every one of which indubitably cause macroscopic bone deformities and anomalous mechanical performance in XLH/Hyp. Also, we show the partnership of osteocytes and their cellular processes to the mineralization pattern.Insoluble amyloid fibrils formed by self-assembly of amyloidogenic regions of protein
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