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All patients had been suspected of having idiopathic pulmonary hemosiderosis or interstitial lung disease at various other hospitals. Them all obtained treatment with corticosteroid before admission, nevertheless the symptoms did not enhance. Furthermore, all customers transported compound heterozygous mutations (c.80AG, c.609GA) in MMACHC and improved substantially after being addressed for cblC deficiency and PAH. CONCLUSIONS CblC deficiency should be
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