https://www.selleckchem.com/pe....ptide/angiotensin-ii
studies would be useful to support this outcome and better guide clinical antibiotic prescribing practices. Accumulation of excessive extracellular matrix (ECM) and aberrant transforming growth factor β (TGF-β) signaling pathway function can be potential therapeutic targets for keloid treatment. In this study, we examined the antifibrotic effect of metformin as a suppressor of TGF-β signaling pathways in human dermal fibroblasts (HDFs) and keloid spheroids. Human dermal fibroblasts were stimulated with TGF-β (10 ng/m
Like
Comment
Share
