https://www.selleckchem.com/pr....oducts/gsk1016790a.h
Additionally, significant effects were observed regarding DNA damage and histone acetylation levels after the two periods of BPA exposure in cells exposed to the toxicant the percentage of DNA repair foci (formed by the co-localization of γH2AX and 53BP1) increased in a dose-dependent manner, whereas the treatment with the toxicant triggered a decrease in the epigenetic marks H3K9ac and H3K27ac. Our in vitro results reveal that BPA seriously interferes with the process of cardiomyocyte differentiation, which could be related to the
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