https://www.selleckchem.com/pr....oducts/trometamol.ht
Additionally, plants with COP1 mutation or exposed to higher light intensity were more sensitive to salt stress. Collectively, our results demonstrate that long-term treatment of ABA leads to activation of COP1 in a light intensity-dependent manner for GLK1 degradation to suppress chloroplast development, which we propose to constitute a mechanism of balancing normal growth and stress responses upon the long-term abiotic stress.We previously showed that femur fracture in mice caused a reduction in bone volume at distant skeletal site