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Triploidy of maternal origin was present in 32 cases (69.6%) and was related to asymmetrical growth limitation, a thin placenta, and low pregnancy-associated plasma necessary protein A and free beta-human chorionic gonadotrophin (β-hCG) levels. Triploidy of paternal source was present in 14 cases (30.4%) and ended up being involving a heightened nuchal translucency, placental molar changes, and a higher no-cost β-hCG. Prospective prediction of the parental origin for the triploidy
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