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Our previous study revealed a defect when you look at the autophagic flux in the lung cells of neonatal rats with hyperoxia‑induced bronchopulmonary dysplasia (BPD), but the main method remains unidentified. More over, you will find few innovative treatments that will completely alter the span of BPD. The present study examined the phrase of Syntaxin 17 (STX17), a protein necessary for autophagosome‑lysosome binding, in alveolar kind II (AT‑II) epithelial cells of neo