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In specific, we highlight current investigations that have elucidated the share of retromer dysfunction to distinct measures of tauopathy such as tau hyperphosphorylation, aggregation, and impaired cognition and behavior. Finally, we talk about the prospective advantageous asset of focusing on retromer for modifying disease burden and determine important considerations with such a method moving toward clinical translation.The mitochondrial pyruvate carrier (MPC) is the access point for the
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