https://www.selleckchem.com/products/cq31.html
Restoring miR-369 reversed the hyperphosphorylation of tau and downregulated Fyn and SRPK2. Additionally, miR-369 was shown to target the 3'UTRs of Fyn and SRPK2 to regulate their expression levels. Conclusion Loss of miR-369 promotes tau phosphorylation by targeting the Fyn and SRPK2 signaling pathways in AD mice, and supplementation with miR-369 might be a valuable option for AD therapeutic studies. Copyright © 2020 Yao, Xian, Fang, Fan and Li.Modality-invariant categorical representations, i.e., shared representation, is thought to play
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