https://pfkfbsignaling.com/ind....ex.php/recognition-a
We investigated the intracellular metabolic mechanisms driving oxLDL-induced trained immunity in personal major monocytes and noticed concomitant upregulation of glycolytic task and air consumption. In two individual cohorts of healthy volunteers, we assessed the effect of hereditary difference in glycolytic genes on the education capacity of monocytes and found that alternatives mapped to glycolytic enzymes PFKFB3 and PFKP influenced trained immunity by oxLDL. Subsequent functional
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