https://bix2189inhibitor.com/t....he-effects-involving
In this study, we unearthed that a fresh miRNA, miR-101, could suppress FHV-1 replication. FHV-1 disease upregulated the expression degree of miR-101 in a cGAS-dependent manner. Furthermore, miR-101 could significantly enhance type I interferon antiviral signaling by targeting suppressor of cytokine signaling 5 (SOCS5), an adverse regulator regarding the JAK-STAT path. Also, knockdown of cellular SOCS5 also suppressed FHV-1 replication due to the enhancement of IFN-I-induc
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