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LOC100506178 substantially induced, while miR-214-5p suppressed the BMP2-induced osteogenic differentiation of hBMSCs. Mechanistically, LOC100506178 ended up being directly bound to miR-214-5p and miR-214-5p focused the 3'-untranslated region of BMP2 to adversely manage its expression. In closing, our data indicate a novel molecular pathway LOC100506178/miR-214-5p/BMP2 in terms of hBMSCs differentiation into osteoblasts, which could facilitate bone anabolism. ©2020 Li et al
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