https://www.selleckchem.com/products/AZD0530.html
007, P = .024) and HOMA-insulin resistance (IR) (β = .508, P = .03). Allopurinol treatment significantly reduced MAGE (4.16 vs 4.65 mmol/L, P less then .001), SDBG (0.99 vs 1.36 mmol/L, P less then .001) and HOMA-IR (2.26 vs 3.01, P less then .001) in gout patients. Conclusion Blood glucose fluctuation increased even in the stage of normal glucose tolerance among gout patients. Blood glucose fluctuations in gout patients were associated with the level of serum uric acid and allopurinol could decrease blood glucose fluctuation as well as
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