https://www.selleckchem.com/pr....oducts/nibr-ltsi.htm
ion by attenuating the NF-κB signaling pathway and rescued osteoporosis caused by mechanical unloading in an HU mouse model . In this research, we demonstrated that Exosomes derived from CMS-treated BMSCs inhibited osteoclastogenesis by attenuating NF-κB signaling pathway activity and ameliorated bone loss caused by mechanical unloading in an HU mouse model, providing new insights into intercellular communication between osteoblasts and osteoclasts under mechanical loading. In this research, we demonstrated that Exosomes derived from
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