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Sickle cell disease (SCD), the most common serious monogenic infection in the field, is renowned for the characteristic vaso-occlusive crises that cause great suffering and degradation of health of these clients. In 1949, the discovery associated with the abnormal sickle-cell hemoglobin necessary protein (HbS) β-globin chain revealed a mutation where glutamic acid is changed with a valine (β6Glu→Val). Out of this breakthrough emerged the pathophysiological device based on the
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