https://www.selleckchem.com/products/CHIR-258.html
The pathological DUA properties were improved by M-MSC treatment in a dose-dependent manner, with the 1000K group producing the best efficacy. Histological analysis revealed that M-MSC therapy reduced CBI-induced injuries including bladder fibrosis, muscular loss, and apoptosis. Transplanted M-MSCs mainly engrafted as vimentin and NG2 positive pericytes rather than myocytes, leading to increased angiogenesis in the CBI bladder. Transcriptomes of the CBI-injured bladders were characterized by the complement system, inflammatory, and ion
Like
Comment
Share
