https://phenanthrolinemonohydr....ate.com/wellbeing-be
In the present research, we aim to explore the part and mechanisms of IL-22 in renal tubular cells inflammation and fibrosis caused by TGF-β1. HK-2 cells had been treated with TGF-β1 in the presence of IL-22 or the Notch pathway inhibitor dibenzazepine (DBZ) for 48 h. Collagen I (Col I), fibronectin (FN), α-smooth muscle mass actin (α-SMA), vimentin and E-Cadherin were detected by western blot, proinflammatory aspects (TNF-α, IL-6) and chemokines (MCP-1, RANTES) had been
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