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We've formerly identified that treatment using the CCR1 ligand CCL3 inhibits the response to CXCL12 in MM cellular outlines, suggesting that CCL3/CCR1 signalling may allow egress of MM Computer through the BM. Right here, we demonstrated that CCR1 expression had been a completely independent prognostic indicator in newly diagnosed MM customers. Moreover, we indicated that CCR1 is a crucial motorist of dissemination in vivo, with
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