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01) and myoglobin (p less then 0.0001) levels decreased in the E + UCII group compared to control. Additionally, UCII supplementation caused significant increases in antioxidant enzyme activities [SOD (p less then 0.01) and GSH-Px (p less then 0.05)] and decreases in malondialdehyde (MDA) and tumor necrosis factor (TNF-α) levels (p less then 0.001). Muscle lipogenic protein (SREBP-1c, ACLY, LXR, and FAS) levels were lower in the E + UCII group than in other groups. In addition, UCII supplementation decreased muscle MAFbx, MuRF-1, myosta