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We further demonstrated that activation of NADPH oxidases (NOX) is the major signaling pathway downstream of Ang II-AT1R leading to stimulation of ClC-K2. Remedy for newly isolated gathering ducts with Ang II resulted in production of reactive oxygen species for a passing fancy timescale as solitary station ClC-K2 activation. Overall, we propose that Ang II-dependent regulation of ClC-K2 in intercalated cells is instrumental for stimulation of Cl- reabsorption becau
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