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BACKGROUND AND AIMS Genetic factors associated with non-alcoholic fatty liver disease (NAFLD) remain incompletely understood. To date, most GWAS studies have adopted radiologically assessed hepatic triglyceride content as reference phenotype and so cannot address steatohepatitis or fibrosis. We describe a genome-wide association study (GWAS) encompassing the full spectrum of histologically characterized NAFLD. METHODS The GWAS involved 1483 European NAFLD cases and 17781 genetically-matched population controls. A replication cohort of 559
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