https://www.selleckchem.com/products/blu-554.html
Exposure to particulate matter (PM) air pollution is associated with adverse health outcomes in children with cystic fibrosis (CF). Airway macrophages (AM) phagocytose and retain inhaled PM in vivo, and the area of carbon in AM reflects both inhaled PM dose and phagocytic function. Since airway prostaglandin-E (PGE ) is increased in CF, and PGE suppresses AM phagocytosis, we sought evidence for PGE -mediated suppression of AM phagocytosis of inhaled carbonaceous PM in CF. After informed consent, urine was obtained from 20 controls and 2
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