https://www.selleckchem.com/products/c25-140.html
The dual mechanisms of increased mitochondrial respiration and enterohepatic bile acid recirculation due to improvement of ER-mitochondria calcium homeostasis with hepatic HAX-1 inactivation suggest that this may be a potential therapeutic target for metabolic disease intervention. Published under license by The American Society for Biochemistry and Molecular Biology, Inc.Excitatory amino acid transporters (EAATs) represent a protein family that is an emerging drug target with great therapeutic potential for managing central nervous sys
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