https://www.selleckchem.com/pr....oducts/rhosin-hydroc
Also, GLI1 knockdown increased expression of thyroid-specific proteins. Restoration of thyroid-specific proteins increased RAI uptake and I-131-mediated cytotoxic effect. Treatment with GANT61 also increased expression of endogenous NIS. Targeting GLI1 can be a potential strategy with redifferentiation for restoring RAI avidity in dedifferentiated thyroid cancers. Measuring quality of care indicators is important for clinicians and decision making in health care to improve patient outcomes. The primary objective was to iden
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