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Silencing JAK2 expression inhibited p-STAT3 protein appearance and β-glucan-induced IL-1β release. And overexpression of JAK2 further promoted β-glucan-mediated p-STAT3 necessary protein and IL-1β productions. These results recommended that β-glucan-induced CXCL8, CXCL11, and IL-1β secretions in rabbit KC might be mixed up in inflammatory reaction of T. mentagrophytes infected rabbit dorsal epidermis. Nevertheless, only IL-1β secretion ended up being promoted because of
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