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We initially evaluated the focus of GSH and cysteine in body organs of old mice; then, the consequence associated with the administration associated with N-butanoyl GSH derivative (GSH-C4) regarding the response of aged mice contaminated with influenza A PR8/H1N1 virus ended up being studied through the dedication of GSH focus in body organs, lung viral titer, IgA and IgG1/IgG2a manufacturing, and Th1/Th2 cytokine profile. Old mice had reduced GSH than young mice in body organs. Also the gene
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