https://www.selleckchem.com/products/ars-853.html
Overexpression of Hsp70 in H9c2 myocardial cells suppressed LPS-induced apoptosis, while inhibition of autophagy with 3-Ma also decreased LPS-induced H9c2 cell apoptosis, suggesting that the protective effect of Hsp70 in sepsis-induced cardiomyopathy was related to autophagy regulation. Overall, these results suggested that Hsp70 protected against sepsis-induced cardiac impairment by attenuating sepsis-induced autophagy. Overall, these results suggested that Hsp70 protected against sepsis-induced cardiac impairment by attenuating sepsis