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CONCLUSIONS mCD36 plays an important role in atherogenesis. With regards to ABI, mCD36 performed robustly as a marker of ATH. Also, it may stratify subjects inside the 'ABI-uncompromised group' commensurate with regards to conventional clinico-pathological ATH danger predisposition. Complement system proteins, their regulators and endpoint effector complex significantly advertise cyst growth by upregulation of oncogenic growth aspects, activation of mitogenic signalling paths and breakage of typical mobile