https://www.selleckchem.com/pr....oducts/crenolanib-cp
Thus, NO produced endogenously during RSV persistence was not enough to control virus replication, although macrophage activation through phagocytosis inhibited replication of the persistent viral genome. In contrast, the NO donor SNAP increased viral genome replication, at least partially by altering the cell cycle, indicating that both sources of NO were not bioequivalent. Keywords cell cycle; endogenous nitric oxide; exogenous nitric oxide; nitric oxide donor; respiratory syncytial virus; viral persistence.Marek's diseas
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