https://www.selleckchem.com/pr....oducts/telotristat-e
05). From a mechanistic perspective, the results showed that the PI3K-AKT signaling pathway was inhibited after overexpression of SFTA1P in NSCLC. Taken together, this work supported that SFTA1P may play a suppressing role in the tumorigenesis of NSCLC by modulating PI3K-AKT signaling pathway to influence cell cycle, which provides a potential and prospective biomarker for NSCLC.Although tripartite motif containing 27 (TRIM27) protein has been implicated in the progression of many cancer types, its role in
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