https://www.selleckchem.com/pr....oducts/gefitinib-bas
Subsequently, NUCB2 was identified as a target gene of miR-335-5p. Additionally, it was confirmed that NUCB2 functioned as an oncogene in LUAD. Rescue assays indicated that LUAD progression inhibited by FTX knockdown could be restored by NUCB2 up-regulation. Conclusion FTX played an oncogenic role in LUAD and contributed to cancer development via targeting miR-335-5p/NUCB2 axis. © The Author(s) 2020.Background The monitoring and management of blood glucose concentration are standard practices in critical settings as hyp
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