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flecting changes in the underlying processes in the transplant. These data suggest that differential serine hydrolase pathways may be active in subclinical versus clinical rejection which requires further exploration in larger patient cohorts. Although this study focused on PR3/PRTN3, this does not preclude the possibility that other enzymes may play critical roles in the rejection process.Faced with chronic inflammation for number of years, unable to work and mostly bed bound, this fifty-two year old woman decided to look elsewhere for
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