https://www.selleckchem.com/pr....oducts/4-phenylbutyr
The TargetScan analysis showed NF-kB1 to be the target of miR-322. Additionally, NF-kB1 was remarkably upregulated in all the breast cancer cells. However, miR-322 overexpression resulted in depletion of NF-kB1 expression in MCF7 cells. Silencing of NF-kB1 also decreased the proliferation rate and colony formation of the MCF7 cells. CONCLUSION To conclude, miR-322 may exhibit therapeutic implications in breast cancer treatment and warrants further investigation.PURPOSE This study was conducted to assess the impact of
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