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esidues in vivo The priC mutation completely abolished function. The priA mutations had no effect by themselves. They did, however, display modest phenotypes in a priB-null strain. These phenotypes were partially suppressed by SSB overproduction. These studies give us further insight into the reactions needed for replication restart.Pro-inflammatory cytokine and chemokines genes drive prostate cancer progression and metastasis molecular mechanism update and the science that underlies racial disparity. comprehensive review article. Isaac J
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