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The enriched DEGs had been mainly associated with the cell pattern, spliceosome, and Toll-like receptor (TLR) signalling pathways. Eventually, the replication of EV71 was dramatically inhibited by TLR2 heterodimers. Our conclusions suggest that AS activities induced by EV71 increase the transcriptomic diversity of HTECs in response to EV71 illness. Furthermore, TLR2 heterodimers possess possible to safeguard HTECs against EV71.The abdominal microbiota is a
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