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To analyze this subject, we employed CRISPR/Cas9 to generate cyp11c1 (11β-hydroxylase) mutant zebrafish outlines. Our study confirms recently published results from an unusual cyp11c1-/- mutant zebrafish range, also reports book facets of the phenotype due to loss of Cyp11c1 function. We report that Cyp11c1-deficient zebrafish display predominantly female secondary sex qualities, but may possess either ovaries or testes. Moreover, we noticed that cyp11c1-/- mutant male zebrafish are profou
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