https://www.selleckchem.com/CDK.html
In vivo assay demonstrated that antagomiR directed against miR-206 restored Th17/Treg balance during the development of OA. MiR-206 contributed to the progression of OA by modulating Th17/Treg imbalance, suggesting that miR-206 inhibition might be a promising therapeutic strategy for the treatment of OA. MiR-206 contributed to the progression of OA by modulating Th17/Treg imbalance, suggesting that miR-206 inhibition might be a promising therapeutic strategy for the treatment of OA. Psychological factors may impact recovery in patients undergoing tr
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