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Vitro studies suggested aggregate formations might be involved in the pathogenesis of the disease. Our results expanded the phenotypes of ACTN2-related myopathy and provided helpful information to clarify the molecular mechanisms. Our results expanded the phenotypes of ACTN2-related myopathy and provided helpful information to clarify the molecular mechanisms. Current theories assume that retrograde memory deficits for semantic information in amnestic mild cognitive impairment (aMCI) are temporally graded and partially sparing most remo
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