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Overexpression of LINC00494 elevated NFκB1 expression and enhanced cell migration, invasion and tumorigenesis, but additional overexpression of FBXO32 interfered with the tumorgenicity of ovarian cancer cells and . Our work demonstrated that LINC00494 promoted ovarian cancer progression by modulating FBXO32 binding with the transcription factor NFκB1. These results provided new insight into the mechanism of ovarian cancer pathogenesis and suggested new therapeutic targets. Our work demonstrated that LINC00494 promoted ovarian cancer prog
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