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Also, FcγRs exert a vital part in terms of crosslinking the natural and adaptive workhorses of immunity. Ablation of just one among these receptors might favorably or adversely influence the immune reaction. Extremely recently, we unearthed that FcRγ derived from normal cytotoxicity triggering receptor 1 (NCR1) curtails CD8+ T cellular expansion and therefore converts an acute viral infection into a chronic one. Such a finding opens a new opportunity
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