https://www.selleckchem.com/pr....oducts/NVP-BHG712.ht
Moreover, CaMKII overexpression enhanced iohexol-induced mPTP opening, mitochondrial damage and renal tubular epithelial cells apoptosis. These findings first identified the novel role of CaMKII in iohexol-induced tubular cells apoptosis and delineated the CaMKII-CypD/mPTP pathway during contrast-induced tubular cell damage. Hence, these results could provide a new strategy for CIN protection.Ubiquitination is one of the most important post-translational modifications which involves in many biological processes. Because mass spectrom
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