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Clinical Trials, NCT02893852. (https//clinicaltrials.gov/ct2/show/NCT02893852). Clinical Trials, NCT02893852. (https//clinicaltrials.gov/ct2/show/NCT02893852).Supraphysiological shear stress and surface-contact are recognized as driving mechanisms of platelet activation (PA) in blood contacting devices (BCDs). However, the competing role of these mechanisms in triggering thrombogenic events is poorly understood. Here, we characterized the dynamics of PA in response to the combined effect of shear stress and material exposure. Human plate