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© The Author(s) 2020.Patients with rheumatoid arthritis (RA) develop attributes of accelerated ageing, including immunosenescence. These changes feature diminished thymic functionality, development of late-differentiated effector T cells, increased telomeric attrition, and extortionate production of cytokines (senescence-associated secretory phenotype). The development of RA was linked to the early growth of age-related co-morbidities, inc
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