https://www.selleckchem.com/pr....oducts/tasquinimod.h
These lesions were alleviated in TCE/CTSLi positive mice. These results provide the first evidence that in TCE-induced immune kidney injury, intracellular complement in podocytes can be over-activated by CTSL and aggravates podocytes injury, thereby damaging glomerular filtration function. Intracellular complement activation and cathepsin L in podocytes may be a potential target for treating immune kidney injury induced by TCE.The epidermal growth factor receptor tyrosine kinase inhibitors (EGFR TKIs) have been approved for non-smal
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