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Endocannabinoid system activity plays a role in the homeostatic protection against aging and therefore may counteract the development of brain ageing. The cannabinoid type 1 (CB1) receptor activity diminishes with the aging process in the brain, which impairs neuronal network stability and cognitive functions. Nonetheless, the underlying mechanisms that link CB1 task and memory decline continue to be unknown. Mitochondrial activity profoundly affects neuronal purpose, therefore age-
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