https://www.selleckchem.com/pr....oducts/mi-3-menin-ml
ted these responses. The predominance of the M2-like phenotype likely occurred due to the increased demand for efferocytosis. However, M2 macrophage activity was ineffective, resulting in emphysema development and worsening of symptoms.[This corrects the article DOI 10.1371/journal.pone.0225285.].The SOD3 variant, SOD3R213G, results from substitution of arginine to glycine at amino acid 213 (R213G) in its heparin binding domain (HBD) and is a common genetic variant, reported to be associated with ischemic heart disease.
Like
Comment
Share
