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The purpose of the current study would be to research anti-tumor effectiveness for the chimeric anti-GD2 antibody (Ab) dinutuximab beta against GBM. METHODS Expression degrees of GD2 and complement regulatory proteins (CRP; CD46, CD55 and CD59) on well-known and newly founded main tumor originated GBM cell lines had been examined by movement cytometry. Ab-dependent mobile (ADCC) and complement-dependent cytotoxicity (CDC) mediated by dinutuximab beta against GBM cells had been determined by a
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