https://www.selleckchem.com/products/tl13-112.html
We previously reported that treatment with chondroitin sulfate from sturgeon bone (CSS promoted anti-apoptotic activity in hydrogen peroxide (H2O2)-treated chondrocytes and had a protective effect on mitochondria. It is known that cells can repair damaged mitochondria through autophagy, thus inhibiting the development of apoptosis. Therefore, it is reasonable to speculate that CSSB treatment may inhibit chondrocyte apoptosis via regulation of autophagy. We observed the mitochondrial morphology of chondrocytes treated with different d
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