https://www.selleckchem.com/products/zeocin.html
IL-1β promoted the apoptosis and ECM degradation in chondrocytes, while circ_0110251 overexpression relieved the effects. Circ_0110251 functioned as the sponge for miR-3189-3p and miR-3189-3p overexpression reversed the effect of circ_0110251 on IL-1β-induced chondrocyte damage. Additionally, SPRY1 served as the target gene of miR-3189-3p. MiR-3189-3p inhibition ameliorated IL-1β-induced chondrocyte apoptosis and ECM degradation, while SPRY1 silencing rescued the impacts. Circ_0110251 protected chondrocytes from IL-1β-induced apoptosis a
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