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The retinoblastoma group of pocket proteins (pRBs), made up of Rb1, p107, and p130 are unfavorable regulators of cell-cycle progression. The deletion of every individual pRB in the auditory system causes locks cells' (HCs) and encouraging cells' (SCs) expansion to different extents. Nonetheless, opening their mixed role in the internal ear through conditional or complete knockout practices is limited by the early death associated with triple knockout. In quiescent cel
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